He's real!. He just needs a teaching position at a university.... of GENE by CHE regulates Reactin and Arpa/ 3 interaction and inhibits cell migration J! { s}:
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of GENE by CHE regulates Reactin and Arpa/ 3 interaction and inhibits cell migration
J! { s}: Xavier, (Xavier, chaosphere/ l " Restarter, RH (Restarter, Raphael Hi“ " Stomacher, M (Bloomaster, Margit)“ 1; Stumpy, M (stumpy,
Maria)“ r, Himmel, M (Himmel, Mirko/ , Morgan, (Morgan, Reginald G.}[ 3"”; Fernandez, MP (Fernandez, Mariannas/ an , Wang, CA (Wang,
conans T Gasman, A (Osman, T Mighta, y (Mighta, T Hjertet, RA (Hjertet, Ruth ail T Etching's, L (Etching's, }[ I "
Hofmann, A (Hofmann, Andreas/ , Linden 3 [Linden Stefania]; Niggel, AA (Niggel, Angelika A} 1' 3"“; Germen, women, Christoph 3. 1”]
Source: SCIENTIFIC REPORTS Volume: 2 Arther Number: 241 DUI: " : JAN
Times Cited: 2 (ttom Web of Science)
Cited References: 88 [view l Citation Map
Abstract: GENE (synonyms: coroner , coroner 3} functions in the reorganization ofthe actin network and is implicated in cellular processes like
protrusion formation, secretion, migration and invasion. We demonstrate that GENE is a binding partner and substrate offroadin mass GEE, which
GENE at 3463 in its Crimminal coiled coil domain. GENE loses the GENE ability to inhibit actin
polymerization, to bundle Reactin, andno binded the complex. As a consequence, mutant GENE changes the morphology ofthe Reactin
network in the front . Our data imply that of GENE is involved in the modulation ofthe local morphology of
complex actin structures inhibits cell migration.
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Submitted: 02/02/2013
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#1 - martycamp (02/02/2013) [-]
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